[Editor's Note: JP-8 jet fuel has been found in every chemtrails analysis I've ever read of, including one of the first analysis of chemtrails obtained by William Thomas and Erminia Cassani in April of 1999. Ken Welsh' recent posting on chemtrails also pointed out that the JP-8 was modified with the addition of a highly toxic-and banned-pesticide just a few years before the chemtrails spraying operations began in earnest in 1998 under the rubric of 'retarding the flammability' of jet fuel in the event of a plane crash (apparently a non-problem that supposedly required a 'solution').

Of course, the reason for the entire chemtrail spraying operation is to compromise and insidiously destroy the body's immune system so when the domestic 'terrorists' release their weaponized bio-plagues, we're expected to die by the millions-on cue and in quick order. But don't get too excited, because it's not going to happen exactly as the traitors have envisioned it.

In the same manner that a method for dispersing chemtrails and the influx of Sylphs to neutralize chemtrails has been realized in due course, higher dimensional forces are hard at work to help humanity prevent the long predicted "pandemics" of Bird Flu, or the deadly strain of Spanish Flu of 1918, etc. from wiping us off the face of the earth. Of course, these forces work their magic with the assistance of humans who are most suited to the task and are willing to step up to the plate. You've read about a few of these folks right here at http://educate-yourself.org.

There is a Universal Law in effect that Sir Isaac Newton claimed as his own, but in reality it was simply rediscovered and given a modern title. This rule of Nature states that : for every action, there is an OPPOSITE and EQUAL reaction. That means that whenever the Dark Side mounts a huge, worldwide destructive campaign to kill and destroy, an equally forceful, but countervailing, campaign is generated by the Light Side to thwart that effort.

So resist the impulse to give way to anxiety, fear, melancholy or depression when you read articles such as this which are merely describing the mechanics of the Dark side agenda. You're only getting HALF of the story. The other side of the story you won't find out about until AFTER it's happened, but rest assured- it will happen. There IS a solution to every problem and it can be no other way-it's a Law. ..Ken].

Various authors
http://educate-yourself.org/cn/jp8jetfueltoxicity31dec05.shtml
December 31, 2005

JP8+100 Jet Fuel Toxicity: Proteomic Analysis
Authors: Frank A. Witzmann; INDIANA UNIV AT BLOOMINGTON SCHOOL OF MEDICINE

Abstract: This final technical report describes the results of experiments that were undertaken to analyze the effect of JP8 jet fuel exposure by aerosol/vapor on quantitative and qualitative gene expression in rodent tissues. The stated objectives were to

1) generate gene expression databases for some of the major rodent target tissues,
2) identify as many of the affected gene products as possible, and
3) apply the observed molecular alterations to elucidating JP8's multifaceted toxicity.

As a result of our efforts we have determined that both acute and chronic JP8 exposure significantly alters protein expression in a range of target organs, even after a period of recovery, and that these changes correspond to histological observations in those organs. Furthermore, the protein alterations observed in rats and mice are suggestive of potential hazard when extrapolated to humans.

Limitations: APPROVED FOR PUBLIC RELEASE
Description: Final rept. 1 Sep 1999-31 Mar 2003
Pages: 22
Report Date: 30 APR 2003
Report number: A890714
Price: $21.95 (17% savings) - Shipping terms

No need for EDB, mark. They have lots of poisonous goodies in there. Try this: http://www.desc.dla.mil/DocumentRepository/t83133e.pdf

•Kerosene-type aviation turbine fuel
•2,6-di-tert-butyl-4-methylphenol (antioxidant) [there are 5 other choices for antioxidant additive formulations--this is the first one]
•N,N'disalycylidene-1,2-propanediamine (metal deactivator)
•Stadis 450 (static dissipator)
•[compound not specified, possibly DCI-4A](corrosion inhibitor/lubricity improver)
•[compound not specified, possibly Di-EGME or Prist](fuel system icing inhibitor)
•SPEC AID 8Q462 or AeroShell Performance Additive 101 (thermal stability improver)

This list comes from Chevron: http://www.chevron.com/prodserv/fuels/bulletin/aviationfuel/4_at_fuel_comp.shtm

And here is another list from http://usapc.army.mil/miscellaneous/JP8HazardsStudy.doc

quote:

JP-8 contains three additives: 1) the icing inhibitor diethylene glycolmonomethyl ether (DiEGME), 0.1% v/v; 2) the anti-static compound Stadis 450, 2 mg/L; and 3) the corrosion inhibitor DCI-4A, 15 mg/L (Allen et al., 2001). The possible toxicity of these individual additives and possible additive or synergistic toxicity with hydrocarbon constituents of the parent fuel has been only minimally researched. JP-8 (100), a new formulation being introduced for use by the USAF, is identical to JP-8 except for the addition of three more performance additives. These additives are 1) the antioxidant butylated hydroxytoluene (BHT), 25 ppm; 2) the metal deactivator (MDA), 3 ppm; and 3) the detergent and dispersant 8Q405, 70 ppm (Kanikkannnan et al., 2001).

From the same paper, here is a summary of known health effects of JP-8:

quote:

24. Summary

1. There is little or no evidence that acute or long-term JP-8 exposures result directly in cancer, serious organic disease, or death in humans.

2. Health effects of JP-8 exposure may be subtle, but persisting, and may occur over prolonged periods of low-dose exposure.

3. Some JP-8 induced health effects may require complex neurobehavioral, proteomic, genomic and metabolomic tests for early identification.

4. There appears to be major differences in JP-8 induced health effects as a function of the duration (acute versus long-term), route of administration (dermal versus respiratory versus oral), and exposure phase (vapor versus aerosol versus raw fuel).

5. From animal studies, it appears that brief exposure to JP-8, in at least aerosol or raw fuel phase, can result in severe and persisting immunosuppression.

6. Animal and in vitro studies indicate that exposure to JP-8 can result in modulation of dermal, pulmonary, hepatic, ocular, and renal systems involved in the metabolism, detoxification, and/or elimination of constituent chemicals of JP-8, as well as other xenobiotics.

7. Results of both human and animal studies would appear to indicate that prolonged "occupational-level" exposure to JP-8 could result in persisting changes in brainstem/cerebellar systems, as well as in neurobehavioral performance capacity.

8. Animal and in vitro studies indicate that acute or long-term exposure to JP-8, at least in aerosol phase, can result in persisting damage to the pulmonary system.

9. Human, animal and in vitro studies indicate that acute or long-term dermal exposure to JP-8 can result in damage to the skin (possible necrosis). There is limited evidence from animal studies that repeated dermal exposure to JP-8 might result in skin cancer.

10. There is limited evidence from animal studies that exposure of females to JP-8 can result in developmental deficits in offspring.

11. There is no direct evidence that JP-8 exposure can result in acute lymphocytic leukemia (ALL). There is minimal evidence that repeated exposure to benzene, at JP-8 occupational levels, can result in development of acute myelogenous leukemia (AML). It is generally unknown if possible immunosuppressive effects of JP-8 exposure, as well as JP-8 induced changes in detoxification systems (i.e., skin, liver, etc.) are correlated with the development of leukemia or other cancers.

[Edited 6 times, lastly by 3T3L1 on 01-19-2002]

Excepts from the “Jet Fuel Forum” http://www.aircareintl.org/guestb_files_jet_fuel/guestbook.html

Extract of "Airports: Deadly Neighbors"
by Charles R. Miller

About the Author: Mr. Miller was formerly a supervisor with a major airline and is currently a director of the Alliance of Residents Concerning O'Hare (AReCO) working on airport environmental issues.

What kinds of health effects may be occurring to the population in your neighborhood can be seen from a report, dated June 20, 1997 to the Georgetown Crime Prevention and Community Council by the Seattle-King County Department of Public Health. Georgetown is an area of Seattle, and surrounds the King County International Airport (Boeing Field), King County, in turn, surrounds greater Seattle. (The Georgetown Council is a sister organization to AReCO and member of US-CAW (United States Citizens Aviation Watch). When comparing hospitalization rates for Georgetown (Zip Code 98108) to those of King and North King Counties, the following, alarming statistics resulted:

a 57% higher asthma rate
a 28% higher pneumonia/influenza rate
a 26% higher respiratory disease rate
an 83% higher pregnancy complication rate
a 50% higher infant mortality rate
genetic diseases are statistically higher
mortality rates are 48% higher for all causes of death: 57% higher for heart disease, a 36% higher cancer death rate with pneumonia and influenza among the top five leading causes
average life expectancy 70.4 years (the same as in many developing nations) compared to Seattle's of 76.0 years.

Did you ever wonder what blows out of a jet airplane? Here is what you'll find in the air around an airport:

Freon 11, Freon 12, Methyl Bromide, Dichloromethane, cis-l,2-Dichloroethylene, 1,1,1-Trichloro-ethane, Carbon Tetrachloride, Benzene, Trichloroethylene, Toluene, Tetrachloroethene, Ethylbenzene, m,p-Xylene, o-Xylene, Styrene, 1,3,5-Trimethyl-benzene, 1,2,4-Trimethylbenzene, o-Dichlorobenzene, Formaldehyde, Acetaldehyde, Acrolein, Acetone, Propinaldehyde, Crotonaldehyde, Isobutylaldehyde, Methyl Ethyl Ketone, Benzaldehyde, Veraldehyde, Hexanaldehyde, Ethyl Alcohol, Acetone, Isopropyl Alcohol, Methyl Ethyl Ketone, Butane, Isopentane, Pentane, Hexane, Butyl Alcohol, Methyl Isobutyl Ketone, n,n-Dimethyl Acetamide, Dimethyl Disulfide, m-Cresol, 4-Ethyl Toulene, n-Heptaldehyde, Octanal, 1,4-Dioxane, Methyl Phenyl Ketone, Vinyl Acetate, Heptane, Phenol, Octane, Anthracene, Dimethylnapthalene (isomers), Flouranthene, 1-methylnaphthalene, 2-methylnaphthalene, Naph-thalene, Phenanthrene, Pyrene , Benzo(a)pyrene, 1-nitropyrene, 1,8-dinitropyrene, 1,3-Butadiene, sulfites, nitrites, nitrogen oxide, nitrogen monoxide, nitrogen dioxide, nitrogen trioxide, nitric acid, sulfur oxides, sulfur dioxide, sulfuric acid, urea, ammonia, carbon monoxide, ozone, particulate matter (PM10, PM2.5).

One aircraft take-off can burn thousands of pounds of fuel.

The pollution from just one, two-minute 747 takeoff is equal to operating 2.4 million lawnmowers simultaneously.

The immunotoxicology of jet fuel:
In the early 1990's the United States Air Force began introducing a new safer, less explosive jet fuel, JP-8. Health complaints by engine mechanics and fuel handlers prompted a systematic effort to study the immunosuppressive potential of JP-8. We are testing the hypothesis that that dermal exposure to JP-8 induces immune suppression. Both DTH in vivo and T cell proliferation in vitro were suppressed following dermal application of JP-8. Suppression results after a single large exposure to JP-8, or following multiple smaller exposures. The effect of JP-8 is selective; doses of jet fuel that completely suppress DTH and T cell proliferation fail to interfere with antibody production. Cytokines and biological response modifiers, especially IL-10 and PGE2 appear to be involved. We are particularly interested in determining the mechanisms underlying JP-8-induced immune suppression. We are particularly interested in determining whether using non-steroidal anti-inflammatory drugs can overcome JP-8-induced immune suppression, thereby reducing the risk factor that jet fuel exposure has on the health status of exposed personnel. Ullrich, S.E. (1999) Dermal application of JP-8 jet fuel induces immune suppression.
Toxicological Sciences 52:61-67. Ullrich, S.E. and Lyons, H.L. (2000) Mechanisms involved in the immunotoxicity induced by dermal application of JP-8 jet fuel.

Toxicological Sciences 58:290-298. sullrich@mdanderson.org

The Federal Agency for Toxic Substances and Disease Registry states that volatile organic compounds in jet exhaust, precisely 1,3-butadiene and benzene pose increased health risks in the exposed populace for leukemia and thyroid cancer.

FUEL WORKERS AND HEMATOLOGIC MALIGNANCIES
by Raphael Metzger, Esq.

Fuel workers are exposed to benzene in gasoline and petroleum solvents. Benzene is used as an additive in gasoline because it prevents engines from knocking. It is present in most gasolines at a concentration of a few percent. Benzene is a natural constituent of crude oil. Ordinary fractional distillation processes are insufficient to remove benzene from refined petroleum distillates. Benzene is therefore present as a contaminant in most petroleum products, although at a much lesser concentration than in gasoline. Benzene is also a major constituent of diesel exhaust.

Benzene is one of the few industrial chemicals that is a known human carcinogen. Exposure to benzene causes leukemia, blood diseases such as aplastic anemia and myelodysplastic syndrome, and also causes hematologic cancers such as leukemias, lymphomas and multiple myeloma.

Benzene is volatile and readily evaporates from gasoline and solvents, exposing workers to benzene vapors. Benzene is also absorbed through the skin. Fuel transport workers are exposed to high levels of benzene in filling and unloading tanker trucks. Service station attendants are exposed to benzene when fueling vehicles. Mechanics and other workers who immerse their hands in gasoline and other petroleum products are exposed to benzene dermally as well as by inhalation. Because of its presence in most petroleum products and its volatility and dermal permeability, benzene presents a substantial health risk to fuel workers such as transport workers, fuel station attendants, and mechanics. Exposure monitoring shows that such workers are often exposed to levels of benzene that are known to cause hematologic disease. Epidemiologic studies show that fuel workers, mechanics and service station attendants have increased risk of hematologic diseases and cancers such as leukemia and lymphoma, as well as kidney cancer. Interesting new research indicates that such workers are not only exposed to benzene at dangerous levels, but that workers in these industries routinely suffer decreased blood cell counts and chromosome damage as they do their jobs.

Recent studies also indicate, contrary to commonly held views, that intermittent exposure to benzene is more dangerous than continuous exposure, because the body produces higher levels of certain toxic metabolites of benzene on intermittent exposure.

BENZENE: Merely One ingredient in Jet Fuel, A HUMAN CARCINOGEN
by Raphael Metzger, Esq.

Benzene is also a known human carcinogen. Benzene causes various types of leukemia, lymphoma, and blood diseases. The first case reports of benzene-induced blood diseases date from 1897. The first report of benzene causing leukemia was published in 1928.

In 1948 the American Petroleum Institute published a toxicological review of benzene, noting that benzene causes leukemia and that the only safe level of exposure to benzene is ZERO ppm (parts per million). The first epidemiologic study of benzene among Pliofilm rubber workers showing significantly increased risks of leukemia was published in 1977. Since then, many epidemiologic studies of benzene have been done, which establish benzene as a cause of various human hematologic cancers and diseases. Among the diseases that have been associated in the literature with benzene are acute myelogenous leukemia (AML), acute lymphocytic leukemia (ALL), chronic myelogenous leukemia (CML), chronic lymphocytic leukemia (CLL), hairy cell leukemia (HCL), non-Hodgkin's lymphoma (NHL), Hodgkin's disease, multiple myeloma, myelodysplastic syndrome (MDS), aplastic anemia, pancytopenia, other cytopenias, myelofibrosis, and polycythemia vera.

The greatest risk is to workers who use various petroleum solvents that contain benzene. The use of benzene as a solvent has been banned in the United States for more than twenty 20 years. Nevertheless, workers who use solvents are still exposed to benzene, because there is some benzene in most petroleum solvents.

. Recent studies from China and Great Britain establish that benzene can cause various hematologic cancers and blood diseases at extraordinarily low doses -- a few ppmy (part per million years). A part per million year (ppmy) of benzene is a cumulative dose of just 1 ppm. OSHA's permissible exposure limit for benzene is 1 ppm. Because benzene can still cause harm at this level, the American Conference of Governmental Industrial Hygienists (ACGIH) has recommended a threshold limit value of .5 ppm, and the National Institute for Occupational Safety and Health (NIOSH) has set a Recommended Exposure Limit of 0.1 ppm.

These are very small amounts of benzene. Only one cup of benzene evaporated in a football field-size building (300' x 165' x 14') produces a 3.3 ppm vapor level, which is 3.3 times the OSHA standard, 6.6 times the ACGIH standard, and 33 times the NIOSH standard!!!

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