[Editor's Note: JP-8 jet fuel has been found in every chemtrails
analysis I've ever read of, including one of the first
analysis of chemtrails obtained by William Thomas and Erminia Cassani in
April of 1999. Ken Welsh' recent posting on chemtrails also pointed
out that the JP-8 was modified with the addition of a highly toxic-and banned-pesticide
just a few years before the chemtrails spraying operations began in earnest
in 1998 under the rubric of 'retarding the flammability' of jet fuel in
the event of a plane crash (apparently a non-problem that supposedly required
Of course, the reason for the entire chemtrail spraying operation is to
compromise and insidiously destroy the body's immune system so when the
domestic 'terrorists' release their weaponized bio-plagues, we're expected
to die by the millions-on cue and in quick order. But don't get too excited,
because it's not going to happen exactly as the traitors have envisioned
In the same manner that a method
for dispersing chemtrails and the influx
of Sylphs to neutralize chemtrails has been realized in due course,
higher dimensional forces are hard at work to help humanity prevent the
long predicted "pandemics" of Bird Flu, or the deadly strain of
Spanish Flu of 1918, etc. from wiping us off the face of the earth. Of course,
these forces work their magic with the assistance of humans who are most
suited to the task and are willing to step up to the plate. You've read
about a few of these folks right here at http://educate-yourself.org.
There is a Universal Law in effect that Sir Isaac Newton claimed
as his own, but in reality it was simply rediscovered and given a modern
title. This rule of Nature states that : for every action, there is
an OPPOSITE and EQUAL reaction. That means that whenever the Dark Side
mounts a huge, worldwide destructive campaign to kill and destroy, an equally
forceful, but countervailing, campaign is generated by the Light Side to
thwart that effort.
So resist the impulse to give way to anxiety, fear, melancholy
or depression when you read articles such as this which are merely describing
the mechanics of the Dark side agenda. You're only getting HALF of the story.
The other side of the story you won't find out about until AFTER it's happened,
but rest assured- it will happen. There IS a solution to every
problem and it can be no other way-it's a Law. ..Ken].
December 31, 2005
JP8+100 Jet Fuel Toxicity: Proteomic Analysis
Authors: Frank A. Witzmann; INDIANA UNIV AT BLOOMINGTON SCHOOL OF MEDICINE
Abstract: This final technical report describes the results of experiments
that were undertaken to analyze the effect of JP8 jet fuel exposure by aerosol/vapor
on quantitative and qualitative gene expression in rodent tissues. The stated
objectives were to
1) generate gene expression databases for some of the major
rodent target tissues,
2) identify as many of the affected gene products as possible, and
3) apply the observed molecular alterations to elucidating JP8's multifaceted
As a result of our efforts we have determined that both acute
and chronic JP8 exposure significantly alters protein expression in a range
of target organs, even after a period of recovery, and that these changes
correspond to histological observations in those organs. Furthermore, the
protein alterations observed in rats and mice are suggestive of potential
hazard when extrapolated to humans.
Limitations: APPROVED FOR PUBLIC RELEASE
Description: Final rept. 1 Sep 1999-31 Mar 2003
Report Date: 30 APR 2003
Report number: A890714
Price: $21.95 (17% savings) - Shipping terms
•Kerosene-type aviation turbine fuel
•2,6-di-tert-butyl-4-methylphenol (antioxidant) [there are 5 other
choices for antioxidant additive formulations--this is the first one]
•N,N'disalycylidene-1,2-propanediamine (metal deactivator)
•Stadis 450 (static dissipator)
•[compound not specified, possibly DCI-4A](corrosion inhibitor/lubricity
•[compound not specified, possibly Di-EGME or Prist](fuel system icing
•SPEC AID 8Q462 or AeroShell Performance Additive 101 (thermal stability
JP-8 contains three additives: 1) the icing inhibitor diethylene
glycolmonomethyl ether (DiEGME), 0.1% v/v; 2) the anti-static compound
Stadis 450, 2 mg/L; and 3) the corrosion inhibitor DCI-4A, 15 mg/L (Allen
et al., 2001). The possible toxicity of these individual additives and
possible additive or synergistic toxicity with hydrocarbon constituents
of the parent fuel has been only minimally researched. JP-8 (100), a new
formulation being introduced for use by the USAF, is identical to JP-8
except for the addition of three more performance additives. These additives
are 1) the antioxidant butylated hydroxytoluene (BHT), 25 ppm; 2) the
metal deactivator (MDA), 3 ppm; and 3) the detergent and dispersant 8Q405,
70 ppm (Kanikkannnan et al., 2001).
From the same paper, here is a summary of known health effects
1. There is little or no evidence that acute or long-term
JP-8 exposures result directly in cancer, serious organic disease, or death
2. Health effects of JP-8 exposure may be subtle, but persisting,
and may occur over prolonged periods of low-dose exposure.
3. Some JP-8 induced health effects may require complex neurobehavioral,
proteomic, genomic and metabolomic tests for early identification.
4. There appears to be major differences in JP-8 induced health
effects as a function of the duration (acute versus long-term), route of
administration (dermal versus respiratory versus oral), and exposure phase
(vapor versus aerosol versus raw fuel).
5. From animal studies, it appears that brief exposure to
JP-8, in at least aerosol or raw fuel phase, can result in severe and persisting
6. Animal and in vitro studies indicate that exposure to JP-8
can result in modulation of dermal, pulmonary, hepatic, ocular, and renal
systems involved in the metabolism, detoxification, and/or elimination of
constituent chemicals of JP-8, as well as other xenobiotics.
7. Results of both human and animal studies would appear to
indicate that prolonged "occupational-level" exposure to JP-8
could result in persisting changes in brainstem/cerebellar systems, as well
as in neurobehavioral performance capacity.
8. Animal and in vitro studies indicate that acute or long-term
exposure to JP-8, at least in aerosol phase, can result in persisting damage
to the pulmonary system.
9. Human, animal and in vitro studies indicate that acute
or long-term dermal exposure to JP-8 can result in damage to the skin (possible
necrosis). There is limited evidence from animal studies that repeated dermal
exposure to JP-8 might result in skin cancer.
10. There is limited evidence from animal studies that exposure
of females to JP-8 can result in developmental deficits in offspring.
11. There is no direct evidence that JP-8 exposure can result
in acute lymphocytic leukemia (ALL). There is minimal evidence that repeated
exposure to benzene, at JP-8 occupational levels, can result in development
of acute myelogenous leukemia (AML). It is generally unknown if possible
immunosuppressive effects of JP-8 exposure, as well as JP-8 induced changes
in detoxification systems (i.e., skin, liver, etc.) are correlated with
the development of leukemia or other cancers.
Extract of "Airports: Deadly Neighbors"
by Charles R. Miller
About the Author: Mr. Miller was formerly a supervisor with a major airline
and is currently a director of the Alliance of Residents Concerning O'Hare
(AReCO) working on airport environmental issues.
What kinds of health effects may be occurring to the population in your
neighborhood can be seen from a report, dated June 20, 1997 to the Georgetown
Crime Prevention and Community Council by the Seattle-King County Department
of Public Health. Georgetown is an area of Seattle, and surrounds the King
County International Airport (Boeing Field), King County, in turn, surrounds
greater Seattle. (The Georgetown Council is a sister organization to AReCO
and member of US-CAW (United States Citizens Aviation Watch). When comparing
hospitalization rates for Georgetown (Zip Code 98108) to those of King and
North King Counties, the following, alarming statistics resulted:
a 57% higher asthma rate
a 28% higher pneumonia/influenza rate
a 26% higher respiratory disease rate
an 83% higher pregnancy complication rate
a 50% higher infant mortality rate
genetic diseases are statistically higher
mortality rates are 48% higher for all causes of death: 57% higher for
heart disease, a 36% higher cancer death rate with pneumonia and influenza
among the top five leading causes
average life expectancy 70.4 years (the same as in many developing nations)
compared to Seattle's of 76.0 years.
Did you ever wonder what blows out of a jet airplane? Here
is what you'll find in the air around an airport:
One aircraft take-off can burn thousands of pounds of fuel.
The pollution from just one,two-minute 747 takeoff
is equal to operating 2.4 million lawnmowers simultaneously.
The immunotoxicology of jet fuel:
In the early 1990's the United States Air Force began introducing a new
safer, less explosive jet fuel, JP-8. Health complaints by engine mechanics
and fuel handlers prompted a systematic effort to study the immunosuppressive
potential of JP-8. We are testing the hypothesis that that dermal exposure
to JP-8 induces immune suppression. Both DTH in vivo and T cell proliferation
in vitro were suppressed following dermal application of JP-8. Suppression
results after a single large exposure to JP-8, or following multiple smaller
exposures. The effect of JP-8 is selective; doses of jet fuel that completely
suppress DTH and T cell proliferation fail to interfere with antibody production.
Cytokines and biological response modifiers, especially IL-10 and PGE2 appear
to be involved. We are particularly interested in determining the mechanisms
underlying JP-8-induced immune suppression. We are particularly interested
in determining whether using non-steroidal anti-inflammatory drugs can overcome
JP-8-induced immune suppression, thereby reducing the risk factor that jet
fuel exposure has on the health status of exposed personnel. Ullrich, S.E.
(1999) Dermal application of JP-8 jet fuel induces immune suppression.
Toxicological Sciences 52:61-67. Ullrich, S.E. and Lyons, H.L. (2000) Mechanisms
involved in the immunotoxicity induced by dermal application of JP-8 jet
The Federal Agency for Toxic Substances and Disease Registry states that
volatile organic compounds in jet exhaust, precisely 1,3-butadiene and benzene
pose increased health risks in the exposed populace for leukemia and thyroid
FUEL WORKERS AND HEMATOLOGIC MALIGNANCIES
by Raphael Metzger, Esq.
Fuel workers are exposed to benzene in gasoline and petroleum solvents.
Benzene is used as an additive in gasoline because it prevents engines from
knocking. It is present in most gasolines at a concentration of a few percent.
Benzene is a natural constituent of crude oil. Ordinary fractional distillation
processes are insufficient to remove benzene from refined petroleum distillates.
Benzene is therefore present as a contaminant in most petroleum products,
although at a much lesser concentration than in gasoline. Benzene is also
a major constituent of diesel exhaust.
Benzene is one of the few industrial chemicals that is a known human carcinogen.
Exposure to benzene causes leukemia, blood diseases such as aplastic anemia
and myelodysplastic syndrome, and also causes hematologic cancers such as
leukemias, lymphomas and multiple myeloma.
Benzene is volatile and readily evaporates from gasoline and solvents, exposing
workers to benzene vapors. Benzene is also absorbed through the skin. Fuel
transport workers are exposed to high levels of benzene in filling and unloading
tanker trucks. Service station attendants are exposed to benzene when fueling
vehicles. Mechanics and other workers who immerse their hands in gasoline
and other petroleum products are exposed to benzene dermally as well as
by inhalation. Because of its presence in most petroleum products and its
volatility and dermal permeability, benzene presents a substantial health
risk to fuel workers such as transport workers, fuel station attendants,
and mechanics. Exposure monitoring shows that such workers are often exposed
to levels of benzene that are known to cause hematologic disease. Epidemiologic
studies show that fuel workers, mechanics and service station attendants
have increased risk of hematologic diseases and cancers such as leukemia
and lymphoma, as well as kidney cancer. Interesting new research indicates
that such workers are not only exposed to benzene at dangerous levels, but
that workers in these industries routinely suffer decreased blood cell counts
and chromosome damage as they do their jobs.
Recent studies also indicate, contrary to commonly held views, that intermittent
exposure to benzene is more dangerous than continuous exposure, because
the body produces higher levels of certain toxic metabolites of benzene
on intermittent exposure.
BENZENE: Merely One ingredient in Jet Fuel, A HUMAN CARCINOGEN
by Raphael Metzger, Esq.
Benzene is also a known human carcinogen. Benzene causes various types of
leukemia, lymphoma, and blood diseases. The first case reports of benzene-induced
blood diseases date from 1897. The first report of benzene causing leukemia
was published in 1928.
In 1948 the American Petroleum Institute published a toxicological review
of benzene, noting that benzene causes leukemia and that the only safe level
of exposure to benzene is ZERO ppm (parts per million). The first epidemiologic
study of benzene among Pliofilm rubber workers showing significantly increased
risks of leukemia was published in 1977. Since then, many epidemiologic
studies of benzene have been done, which establish benzene as a cause of
various human hematologic cancers and diseases. Among the diseases that
have been associated in the literature with benzene are acute myelogenous
leukemia (AML), acute lymphocytic leukemia (ALL), chronic myelogenous leukemia
(CML), chronic lymphocytic leukemia (CLL), hairy cell leukemia (HCL), non-Hodgkin's
lymphoma (NHL), Hodgkin's disease, multiple myeloma, myelodysplastic syndrome
(MDS), aplastic anemia, pancytopenia, other cytopenias, myelofibrosis, and
The greatest risk is to workers who use various petroleum solvents that
contain benzene. The use of benzene as a solvent has been banned in the
United States for more than twenty 20 years. Nevertheless, workers who use
solvents are still exposed to benzene, because there is some benzene in
most petroleum solvents.
. Recent studies from China and Great Britain establish that benzene can
cause various hematologic cancers and blood diseases at extraordinarily
low doses -- a few ppmy (part per million years). A part per million year
(ppmy) of benzene is a cumulative dose of just 1 ppm. OSHA's permissible
exposure limit for benzene is 1 ppm. Because benzene can still cause harm
at this level, the American Conference of Governmental Industrial Hygienists
(ACGIH) has recommended a threshold limit value of .5 ppm, and the National
Institute for Occupational Safety and Health (NIOSH) has set a Recommended
Exposure Limit of 0.1 ppm.
These are very small amounts of benzene. Only one cup of benzene evaporated
in a football field-size building (300' x 165' x 14') produces a 3.3 ppm
vapor level, which is 3.3 times the OSHA standard, 6.6 times the ACGIH standard,
and 33 times the NIOSH standard!!!
All information posted on this web site is
the opinion of the author and is provided for educational purposes only.
It is not to be construed as medical advice. Only a licensed medical doctor
can legally offer medical advice in the United States. Consult the healer
of your choice for medical care and advice.